Indian Journal of Dental Research

SYSTEMATIC REVIEW
Year
: 2020  |  Volume : 31  |  Issue : 2  |  Page : 305--311

Abfraction: Etiopathogenesis, clinical aspect, and diagnostic-treatment modalities: A review


Anand N Badavannavar1, Sneha Ajari2, Krishna U S. Nayak3, Shahnawaz Khijmatgar4 
1 Department of Orthodontics, KLE VK Institute of Dental Sciences, KLE Academy of Higher Education and Research, Belgaum, Karnataka, India
2 Department of Oral Pathology,KLE VK Institute of Dental Sciences, KLE Academy of Higher Education and Research Belgavi, Karnataka, India
3 A.B Shetty Memorial Institute of Dental Sciences, Nitte (Deemed to be University), Mangalore, Karnataka, India
4 Department of Oral Biology and Genomic Studies, A B Shetty Memorial Institute of Dental Sciences, Nitte (Deemed to be University), Mangalore, Karnataka, India

Correspondence Address:
Dr. Shahnawaz Khijmatgar
Department of Oral Biology and Genomic Studies, A B Shetty Memorial Institute of Dental Sciences, Nitte (Deemed to be University), Mangalore, Karnataka - 575 018
India

Background: Abfraction is a loss of tooth structure along the gingival margin and manifests with different clinical appearances. It has multifactorial etiology and may occur due to normal and abnormal tooth function and may also be accompanied by pathological wear, such as abrasion and erosion. The theory behind the abfraction is that the tooth flexure in the cervical area is caused due to occlusal compressive forces and tensile stresses. This results in the fractures in the hydroxyapatite (HA) crystals. It is also caused by the low packing density of the Hunter–Schreger band (HSB) at the cervical area. Unfortunately, there is a lack of evidence regarding the outcome of abfraction with or without intervention. The aim of this review is to collect clinical information from the literature and discuss the etiology, pathogenesis, clinical representation, and management. Also, search databases for clinical studies that describe the role of sclerotic dentine in non-carious cervical lesions (NCCLs) are becoming a clinical challenge. Methods: The literature was searched that described the etiology, pathogenesis, clinical representation, and management of the abfraction lesions. Also, a specific question regarding the formation of sclerotic dentin in the NCCL lesion was described and searched for evidence that challenges etching, bonding, and successfully restoring NCCLs. The databases PUBMED, SCOPUS, MEDLINE, WEB of SCIENCE, and EMBASE were searched using the key terms. The inclusion criteria were the randomized controlled clinical trial, cohort studies, and cross-sectional studies that aimed at determining the role of sclerotic dentine in NCCLs and its effect on etching, bonding. Results: One clinical study was retrieved according to the PRISMA flowchart and PICO format. The longer etching time, total-etch adhesive system, and EDTA pre-treatment of the sclerotic dentin of cervical wedge-shaped defects could improve the bonding strength in lesions like NCCL's. Conclusion: In conclusion, clinical challenges that occur due to NCCLs are better managed by a proper understanding of factors like etiopathogenesis, ultra-structure of enamel, and dentine and their effect on the bonding of restorations of the tooth.


How to cite this article:
Badavannavar AN, Ajari S, Nayak KU, Khijmatgar S. Abfraction: Etiopathogenesis, clinical aspect, and diagnostic-treatment modalities: A review.Indian J Dent Res 2020;31:305-311


How to cite this URL:
Badavannavar AN, Ajari S, Nayak KU, Khijmatgar S. Abfraction: Etiopathogenesis, clinical aspect, and diagnostic-treatment modalities: A review. Indian J Dent Res [serial online] 2020 [cited 2020 Jul 5 ];31:305-311
Available from: http://www.ijdr.in/article.asp?issn=0970-9290;year=2020;volume=31;issue=2;spage=305;epage=311;aulast=Badavannavar;type=0